Translation of "Vascular endothelial cells" in German

The at least one vessel branch is luminally populated preferably with autologous (receiver inherent) vascular endothelial cells.

Many of the cells that demonstrated positive immunostaining were vascular endothelial cells and retinal pigment epithelial cells, as well as other extravascular components.

In vitro, ixazomib affected cell types found in the bone marrow microenvironment including vascular endothelial cells, osteoclasts and osteoblasts.

Although not studied directly with lenvatinib, the mechanism of action (MOA) for hypertension is postulated to be mediated by the inhibition of VEGFR2 in vascular endothelial cells.

The leucocytes circulating in the blood normally exhibit only a low affinity for the vascular endothelial cells which line the blood vessels.

These specific target structures could be utilized in order to apply cytostatics or certain toxins to the vascular endothelial cells of the tumor to a lesser extent to the tumor cells themselves.

To generate bioprinted heart tissue, tissue spheroids of human vascular endothelial cells (HUVECs) and cardiac cells were generated.

In inflamed and vascularized corneas positive immunostaining of JAM-1, JAM-2 and JAM-3 was also found on vascular endothelial cells of newly formed vessels in the stroma.

It is also reported to be expressed in normal breast, including vascular endothelial cells, macrophages, myoepithelial cells, osteosarcomas but not in lymphoid tumors.

While searching for pathogens, they roll along the vascular wall and establish initially only loose contact with vascular wall endothelial cells.

These are e.g., benign or malignant tumors, particularly tumors of epithelial and neuroepithelial origin, metastasization and the abnormal proliferation of vascular endothelial cells (neoangiogenesis).

Angiogenesis is the process that follows valculogenesis, during which new blood vessels develop from pre-existing ones through migration, proliferation and finally three-dimensional re-organisation of vascular endothelial cells.

In addition fibroblasts of the skin, epithelial cells of the thymus and the thyroid gland, glial cells, beta cells of the pancreas as well as vascular endothelial cells may act as non-professional APC.

The term “antigen presenting cell” comprises in particular so-called professional antigen presenting cells such as dendritic cells, monocytes, macrophages, non-professional APC, such as B cells, but also vascular endothelial cells, fibroblasts of the skin, epithelial cells of the thymus or the thyroid gland, glial cells of the brain, beta cells of the pancreas as well as vascular endothelium cells.

In the case of a fresh wound or skin injury to be cared for, inflammatory processes occur within the first 24 hours, which include, in particular, the immigration of diverse inflammation factors (such as, for example, fibronectin) and cells of various types, such as, for example, monocytes, phagocytes, polymorphic cells and macrophages, and ultimately result in the formation of a fibrin matrix and vascular endothelial cells.

Topically and locally applied EPO in accordance with the invention furthermore causes no or substantially no stimulation of vascular or endothelial precursor cells circulating systemically in the bloodstream.

Expression of VEGF receptors that mediate mitogenic responses to the growth factor is largely restricted to vascular endothelial cells.

In the case of a fresh wound or skin injury which is to be treated, inflammatory processes, which encompass, in particular, the ingress of diverse inflammation factors (such as, for example, fibronectin) and cells of various types, such as, for example, monocytes, phagocytes, polymorphic cells and macrophages, take place within the first 24 hours, ultimately resulting in the formation of a fibrin matrix and vascular endothelial cells.

These are, for example, benign or malignant tumours, in particular tumours of epithelial and neuroepithelial origin, formation of metastases and the abnormal proliferation of vascular endothelial cells (neoangiogenesis).

Aim: The proliferation of vascular endothelial cells and ultimately angiogenesis is inhibited by blocking integrin-mediated cell-matrix interaction.

Macrophages, specialized cells in the immune system, produce the growth factor VEGF, which boosts the production and function of GLUT-1, and release it directly at the vascular endothelial cells of the blood-brain barrier.

Research has shown Jiaogulan to decrease free radicals in immune cells, protected biomenbranes from oxidative injury, and improve functioning of vascular endothelial cells.

It has the ability to protect vascular endothelial cells, avoid damage from free radicals, and prevent arteriosclerosis and other cardiovascular diseases from occurring.